Nora Schultz Male bees with an urge to add to the gene pool are exploited by orchids that pose as irresistibly scented exotic females, new research suggests. Sexually deceptive orchids rely for their own reproduction on male insects' drive to copulate. If a male mistakes the flowers for a female insects, his fruitless mating attempts will pollinate the orchid. Some orchid flowers are so exciting to males that they even ejaculate on the flower, but although the visual deception is often superb, what really drives the males wild is the female sex pheromone cocktail that the orchid imitates. Now Nicolas Vereecken and Florian Schiestl at the University of Zürich in Switzerland have discovered a new trick in the orchids' repertoire of sexual lures. When they studied geographic variation in pheromone mixes between 15 different populations of the bee Colletes cunicularius and the orchid that mimics it, Ophrys exaltata, they were surprised to find that the flowers consistently smelled slightly different than the female bees in any given population. "This was not at all what we expected. If the orchids thrive on imitating female bees, the match should be as perfect as possible", says Schiestl. © Copyright Reed Business Information Ltd

Keyword: Sexual Behavior; Chemical Senses (Smell & Taste)
Link ID: 11668 - Posted: 06.24.2010

Linda Geddes They say you are only as old as the woman (or man) you feel – now we might be closer to understanding why. It has been suggested that humans and other vertebrates live longer if they have more social interactions, and now this has been verified – in fruit flies. Chun-Fang Wu and Hongyu Ruan at the University of Iowa in Iowa City studied fruit flies with a genetic mutation that reduces their lifespan by interfering with an enzyme that mops up dangerous free radicals. The same enzyme is implicated in age-related diseases like Alzheimer's and Parkinson's in humans. Mutant flies that shared a home with younger flies, or non-mutants, lived longer and were more mobile than those sharing a home with similar-aged flies. They were also more resistant to the effects of extreme physical exertion, heat and oxidative stress. Impairing the movement or activity of younger flies reduced this effect, suggesting that social interaction with the younger flies through courtship, aggression, or grooming, plays a key role in increasing the lifespan of the older flies. "Social activity is the key," says Wu. Journal reference: Proceedings of the National Academy of Sciences (DOI: 10.1073/pnas.0711127105) © Copyright Reed Business Information Ltd

Keyword: Alzheimers
Link ID: 11667 - Posted: 06.24.2010

By VIRGINIA HEFFERNAN You don’t have to search very hard to find the excruciating online videos known as thinspiration, or thinspo. Photomontages of skeletal women, including some celebrities and models, play all over the Internet, uploaded from the United States, Germany, Holland and elsewhere. These videos are designed to “inspire” viewers — to fortify their ambitions. But exactly which ambitions? To lose weight, presumably. To stop losing weight, possibly. Thinspo videos profess a range of ideologies, often pressing morbid images into double service, as both goads and deterrents to anorexia. Thinspiration videos are a cryptic art with rigid rules, as much a formula as a form. As listless, pounding or archly chipper music plays, still photos of one wraith after another surface and fade. The women are generally solitary and sullen, or entirely faceless. Bony self-portraits, created in bathroom mirrors by anonymous photographers, have faces that have been obscured or cropped out. Many figures in the videos are supine, as in the pervasive hipbone self-portrait, which seems to be shot by a photographer on her back aiming a camera at her abdomen and the waistband of her jeans. A bird’s-eye shot of the thighless legs of a seated figure is also common. The soundtracks to thinspiration videos, some of which feature songs explicitly about starvation, are not subtle. Skeleton, you are my friend. I will sacrifice all I have in life. Bones are beautiful. Hey, baby, can you bleed like me? Filmmakers are reticent with commentary. If they explain their images in any way, it’s with oddly peppy title cards (“Enjoy!” “Thanks for watching!”) or a series of unsigned quotations, compiled as if for a commonplace book. A thinspiration auteur makes her voice heard almost exclusively through these cards, and she sometimes uses them to plead with her audience to go easy on her work or to stay tuned for further thinspo. I’ve never seen a thinspo video with a voice-over or even moving images. Copyright 2008 The New York Times Company

Keyword: Anorexia & Bulimia
Link ID: 11666 - Posted: 06.24.2010

By Ariel David ROME - No one can read our thoughts, for now, but some scientists believe they can at least figure out in what language we do our thinking. Before we utter a single word, experts can gauge our mother tongue and the level of proficiency in other languages by analyzing our brain activity while we read, scientists working with Italy's National Research Council say. For more than a year, a team of scientists experimented on 15 interpreters, revealing what they say were surprising differences in brain activity when the subjects were shown words in their native language and in other languages they spoke. The findings show how differently the brain absorbs and recalls languages learned in early childhood and later in life, said Alice Mado Proverbio, a professor of cognitive electrophysiology at the Milano-Bicocca University in Milan. Proverbio, who led the study, said such research could help doctors communicate with patients suffering from amnesia or diseases that impair speech. It could also be of use one day in questioning refugee applicants or terror suspects to determine their origin, she said. The interpreters who took part in the study were all Italians working for the European Union and translating in English and Italian. © 2008 The Associated Press.

Keyword: Language; Brain imaging
Link ID: 11665 - Posted: 06.24.2010

By MICHAEL TOMASELLO Human beings do not like to think of themselves as animals. It is thus with decidedly mixed feelings that we regard the frequent reports that activities once thought to be uniquely human are also performed by other species: chimpanzees who make and use tools, parrots who use language, ants who teach. Is there anything left? You might think that human beings at least enjoy the advantage of being more generally intelligent. To test this idea, my colleagues and I recently administered an array of cognitive tests — the equivalent of nonverbal I.Q. tests — to adult chimpanzees and orangutans (two of our closest primate relatives) and to 2-year-old human children. As it turned out, the children were not more skillful overall. They performed about the same as the apes on the tests that measured how well they understood the physical world of space, quantities and causality. The children performed better only on tests that measured social skills: social learning, communicating and reading the intentions of others. But such social gifts make all the difference. Imagine a child born alone on a desert island and somehow magically kept alive. What would this child’s cognitive skills look like as an adult — with no one to teach her, no one to imitate, no pre-existing tools, no spoken or written language? She would certainly possess basic skills for dealing with the physical world, but they would not be particularly impressive. She would not invent for herself English, or Arabic numerals, or metal knives, or money. These are the products of collective cognition; they were created by human beings, in effect, putting their heads together. Copyright 2008 The New York Times Company

Keyword: Evolution; Intelligence
Link ID: 11664 - Posted: 06.24.2010

By Rob Stein Facebook, MySpace and other Web sites have unleashed a potent new phenomenon of social networking in cyberspace. But at the same time, a growing body of evidence is suggesting that traditional social networks play a surprisingly powerful and underrecognized role in influencing how people behave. The latest research comes from Nicholas A. Christakis, a medical sociologist at the Harvard Medical School, and James H. Fowler, a political scientist at the University of California at San Diego. The pair reported last summer that obesity appeared to spread from one person to another through social networks, almost like a virus or a fad. In a follow-up to that provocative research, the team has produced similar findings about another major health issue: smoking. In a study published last week in the New England Journal of Medicine, the team found that a person's decision to kick the habit is strongly affected by whether other people in their social network quit -- even people they do not know. And, surprisingly, entire networks of smokers appear to quit virtually simultaneously. Taken together, these studies and others are fueling a growing recognition that many behaviors are swayed by social networks in ways that have not been fully understood. And it may be possible, the researchers say, to harness the power of these networks for many purposes, such as encouraging safe sex, getting more people to exercise or even fighting crime. © 2008 The Washington Post Company

Keyword: Drug Abuse
Link ID: 11663 - Posted: 06.24.2010

NEW drugs may help to enhance people's mental powers (see article). But a study carried out by Pamela Smith, of Radboud University Nijmegen in the Netherlands, and her colleagues suggests a less pharmacological approach can be taken, too. Their work, just published in Psychological Science, argues that simply putting someone into a weak social position impairs his cognitive function. Conversely, “empowering” him, in the dread jargon of sociology, sharpens up his mind. Dr Smith focused on those cognitive processes that help people maintain and pursue their goals in difficult and distracting situations. She suspected that a lack of social power may reduce someone's ability to keep track of information and make plans to achieve his goals. To explore this theory, she carried out three tests. In the first, participants were divided at random into groups of superiors and subordinates. They were told that the superiors would direct and evaluate the subordinates and that this evaluation would determine the subordinates' payment for the experiment. Superiors were paid a fixed amount. The subordinates were then divided into two further groups: powerless and empowered. A sense of powerlessness was instilled, the researchers hoped, by having participants write for several minutes about a time when they were powerless or by asking them to unscramble sets of words including “obey”, “subordinate” and so on to form sentences. The empowered, by contrast, were asked to write about when they had been on top, or to form sentences including “authority”, “dominate” and similar words. © The Economist Newspaper Limited 2008.

Keyword: Emotions
Link ID: 11662 - Posted: 06.24.2010

By DANIEL BERGNER “You want to wear this or this for therapy tomorrow?” Sgt. Shurvon Phillip’s mother asked, holding two shirts in front of him. On one wall of his bedroom hung a poster of a marine staring fiercely, assault rifle in hand and black paint beneath his narrow eyes. Shurvon’s eyes, meanwhile, are wide and soft brown. He sat upright, supported by the tilt of a hospital bed. He cannot speak and can barely emit sound or move any part of his body, and sometimes it’s as if the striking size of his eyes is a desperate attempt to let others understand who he is, to let them see inside his mind, because his brain can carry out so little in the way of communication. Keeping Hope Alive Shurvon’s mother has been his greatest advocate and believer. He gazed at the two shirts and, with excruciating effort and several seconds’ delay, managed to jab his gnarled right hand a few inches toward his choice, a black pullover with writing on the front. White letters declared the man, and a white arrow pointed upward to his head; red letters proclaimed the legend, and a red arrow pointed downward to his groin. Gail Ulerie, Shurvon’s mother, had already received his O.K. — a painstaking raising of his eyebrows — on a pair of jeans. Mostly, Shurvon can answer only yes-or-no questions. The slightly lifted brows, a gesture that stretches his eyes yet wider, signify yes. A slow lowering of his lids indicates no. Now, with tomorrow’s clothes decided, Gail, a Trinidadian-American, reclined Shurvon’s bed for the night. Copyright 2008 The New York Times Company

Keyword: Brain Injury/Concussion
Link ID: 11661 - Posted: 06.24.2010

By KYLA DUNN Behind all the good-natured joking about “senior moments” lies real frustration and fear. How empowering, then, that we can do something to ward off normal, age-related memory loss: exercise. No, it’s not as easy as popping a pill. But as Sue Halpern reports in “Can’t Remember What I Forgot,” even a few brisk walks per week can have a measurable effect. Exercise promotes the birth of new neurons in the very part of the hippocampus (a brain structure crucial to forming new memories) that begins to malfunction with age. Exercise also counters age-related shrinking of the prefrontal cortex, an area involved in concentration and working memory (as in remembering a phone number long enough to dial). Of course, if you’re unwilling to exercise for your health, or even to look good in a bathing suit, perhaps you’re still hoping for an easier solution. The problem, Halpern explains, is that solid, peer-reviewed science has not yet proved that anything else works: not herbal supplements, fish oil, vitamin E, almonds, $400 interactive computer software or even crossword puzzles. (After reading her section on blueberries, however, you’ll want to buy them by the bucketful.) To cut through the hype and confusion, Halpern, whose father had dementia, set out to discover what scientists really know, and how close they are to a drug or therapy that works — either for the pathological memory loss caused by Alzheimer’s, or the “normal” kind that happens inexorably as we age. Her most interesting passages highlight the distinctions between the two. Both ravage the hippocampus; yet a different part of the hippocampus is “broken” in each condition, and different molecules have stopped doing their jobs. “To understand something to the point of being able to fix it you have to get down to the molecular level,” Scott Small, a neurologist at Columbia, tells Halpern. “And that’s where we’re at.” Copyright 2008 The New York Times Company

Keyword: Learning & Memory
Link ID: 11660 - Posted: 06.24.2010

FOR thousands of years, people have sought substances that they hoped would boost their mental powers and their stamina. Leaves, roots and fruit have been chewed, brewed and smoked in a quest to expand the mind. That search continues today, with the difference only that the shamans work in pharmaceutical laboratories rather than forests. If asked why, the shamans reply that they are looking for drugs to treat the effects of Alzheimer's disease, attention-deficit disorder, strokes, and the dementias associated with Parkinson's disease and schizophrenia—and that is the truth. But by creating compounds that benefit the sick, they are offering a mental boost to the healthy, too. Such drugs are known as cognition enhancers. They work on the neural processes that underlie such mental activities as attention, perception, learning, memory, language, planning and decision-making, usually by altering the balance of the chemical neurotransmitters involved in these processes. This week a report* from the Academy of Medical Sciences, a British learned society, says that a large number of such brain-affecting drugs are likely to emerge over the next few decades. Sir Gabriel Horn, a researcher at Cambridge University who chaired the group that produced the report, reckons that scientists are working on more than 600 drugs for neurological disorders. History suggests that most of these will fall by the regulatory wayside, but given their numbers, a fair few are likely to be approved. And although none of the companies working on cognition-enhancing drugs designed to treat illness intends to license them for wider use, that is what is likely to happen—at least going by the growing “off-label” use of existing drugs such as Ritalin (methylphenidate) and Provigil (modafinil) by people who want to pep themselves up. © The Economist Newspaper Limited 2008.

Keyword: Learning & Memory
Link ID: 11659 - Posted: 06.24.2010

She's working productively now, but Cynthia, who prefers not to reveal her last name, struggled for much of her adult life with schizophrenia, a mental illness that can cause confusion, paranoia, and hallucinations. She says one of the scariest symptoms was hearing voices. "Because sometimes the voices could have you to act out, you know in a certain behavior, you know, be explosive, yelling at somebody." Technically, her illness is called schizoaffective disorder because it also includes symptoms of bipolar disorder. Schizophrenia can run in families, as it does in Cynthia's family, but most cases seem to come out of the blue. Psychiatrist Jon McClellan of Seattle Children's Hospital says that's one reason finding a genetic cause of the illness is difficult. "Typically the way it works now–and it's common sense–you start with everybody that has the same syndrome, whether it's schizophrenia or autism or bipolar disorder, and then you try to see what genetic markers they share, with the assumption that because they have the same illness, they must share some similar genetic cause" But schizophrenia does not fit that model. So far, a single genetic cause has been elusive. McClellan and colleagues at the University of Washington, Seattle published a paper in the journal Science explaining a different approach. They examined the entire genome of 150 people with schizophrenia and 268 controls, looking for repeats or deletions–called copy number variants–in small stretches of subjects' DNA. © ScienCentral, 2000-2008

Keyword: Schizophrenia; Genes & Behavior
Link ID: 11658 - Posted: 06.24.2010

Rachel Courtland Does the body make sure we're awake when it's time to eat?PunchstockYour stomach may truly have a mind of its own. A tiny area of the brain may switch sleep schedules to match up with mealtimes. It's been known for a long time that nocturnal creatures such as mice and bats flip their sleep schedules if food is only available during the day. But finding the parts of the brain responsible for the switch has proved difficult. In a paper published today in Science 1, a team led by Clifford Saper from Harvard Medical School in Boston, Massachusetts suggests they have found the region of the brain responsible for the sleep-rhythm adjustment — a clump of cells known as the dorsomedial hypothalamic nucleus (DMH). This region sits close to the area of the brain that manages ordinary circadian responses to light and dark. The study shows that mice lacking a particular gene that acts in the DMH do not adjust to changes in feeding schedule. Reinstating the gene restored the behaviour. But some researchers in the field have serious concerns about the work. “On the face of it, it’s almost the final nail in saying DMH is the pacemaker, but under the surface there are people who strongly disagree,” says neuroscientist Masashi Yanagisawa of University of Texas Southwestern Medical Center in Dallas, who was not involved in the work. © 2008 Nature Publishing Group

Keyword: Biological Rhythms
Link ID: 11657 - Posted: 06.24.2010

Ewen Callaway It can be irritating to get a catchy song stuck in your head. Imagine if the music sounded so real that you were sure it was coming from a stereo, and the tune never went away. This is what it's like to suffer from musical hallucinosis, a mysterious condition that usually strikes elderly people with poor hearing. Ramon Mocellin, a psychiatrist at Royal Melbourne Hospital in Australia who treats patients with the disorder, tells New Scientist about the condition. Are people with the condition mentally ill? Hallucinations, which simply put are perceptions without a stimulus, can be symptoms of mental illness. Auditory hallucinations, in particular hearing voices, are one of the diagnostic criteria of schizophrenia. It is, however, the nature of the hallucinations and the patient's understanding of them that point to the underlying problem. In this group of patients, the experience of hearing music when there is no external source of music is often accompanied by some degree of understanding that these experiences are not "real", that they originate from their own mind. In schizophrenia, or other mental illnesses, hallucinations are experienced as real, in the external world. © Copyright Reed Business Information Ltd.

Keyword: Hearing
Link ID: 11656 - Posted: 06.24.2010

Katharine Sanderson If you thought a high-pitched growl would come only from a tiny dog, you could be in for a nasty surprise. Researchers have shown that it isn’t the fundamental frequency, or pitch, of a growl that humans use to gauge a dog’s size — it’s another acoustic property related to the length of the vocal tract. It was known that within species, the formant — a property of a sound wave related to the length of the vocal tract — is used by animals to assess the size of other animals. But it had never been shown to happen between species. Anna Taylor, a doctoral student at the University of Sussex, Brighton, UK, set out to show that the formant is used between species as a cue for size by seeing how humans respond to growls from different-sized dogs. Her results are published in The Journal of the Acoustical Society of America .1 Taylor visited the homes of more than 100 dogs, armed with nothing but a microphone, a steely stare, and the dog owners' consent. Taylor made the dogs growl defensively by invading the dog's space and staring it in the eyes. She recorded these snarly responses, 30 of which she went to on to manipulate for her experiment. That might sound like an unwise experiment for anyone who values their personal safety. But Taylor says that, as an experienced animal behaviourist, she managed to diffuse any encounters before they turned violent. © 2008 Nature Publishing Group

Keyword: Hearing; Animal Communication
Link ID: 11655 - Posted: 06.24.2010

By GINA KOLATA For years, smokers have been exhorted to take the initiative and quit: use a nicotine patch, chew nicotine gum, take a prescription medication that can help, call a help line, just say no. But a new study finds that stopping is seldom an individual decision. Smokers tend to quit in groups, the study finds, which means smoking cessation programs should work best if they focus on groups rather than individuals. It also means that people may help many more than just themselves by quitting: quitting can have a ripple effect prompting an entire social network to break the habit. The study, by Dr. Nicholas Christakis of Harvard Medical School and James Fowler of the University of California, San Diego, followed thousands of smokers and nonsmokers for 32 years, from 1971 until 2003, studying them as part of a large network of relatives, co-workers, neighbors, friends and friends of friends. It was a time when the percentage of adult smokers in the United States fell to 21 percent from 45 percent. As the investigators watched the smokers and their social networks, they saw what they said was a striking effect — smokers had formed little social clusters and, as the years went by, entire clusters of smokers were stopping en masse. So were clusters of clusters that were only loosely connected. Dr. Christakis described watching the vanishing clusters as like lying on your back in a field, looking up at stars that were burning out. “It’s not like one little star turning off at a time,” he said. “Whole constellations are blinking off at once.” Copyright 2008 The New York Times Company

Keyword: Drug Abuse
Link ID: 11654 - Posted: 06.24.2010

Feeling sad and bad about ourselves is not only unpleasant — it can also be hard on our wallets. Psychology researchers have found that these emotions can cost you three times more for the same item than being in a better mood, as this ScienCentral News video reports. Getting the blues could cost you more of the green. Psychology researchers have found that feeling sad and bad about ourselves can drive us to spend more on purchases. "There are these general hypotheses that if you're just in a negative mood, that that negativity will generalize to everything. But we see that that's not true with sadness," explains Cynthia Cryder, a doctoral candidate in the Department of Social and Decision Sciences at Carnegie Mellon University. "Sad people tend to feel negative about themselves and then increase value of things that aren't associated with themselves like these commodities that they're buying," she says. Cryder and colleagues offered volunteers ten dollars to participate in what were presented as separate experiments that were combined for convenience. Participants first watched either a sad or an unemotional video and wrote an essay designed to reveal how they were feeling about themselves. Then they were asked how much a sports water bottle was worth and to name their price to buy it. © ScienCentral, 2000-2008.

Keyword: Emotions
Link ID: 11653 - Posted: 06.24.2010

Alison Motluk The way humans are conditioned by fearful stimuli is to some extent damped down by the body's own pain-relief system, a study suggests. The finding may shine light on the neural mechanisms behind anxieties, phobias and even post-traumatic stress disorder. Scientists have known for a long time that if you pair an innocuous stimulus (such as a tone) with something aversive (such as a shock to the feet), animals, including humans, will learn to show a "conditioned fear" response. With repetition, the innocuous stimulus alone brings on the fear response. Both the learning and the initiation of this response take place in a part of the brain known as the amygdala. One of the more perplexing features of the conditioned fear response is that, when the dreaded stimulus is something painful, people actually tend to experience less pain the more they are exposed to it. Work in rodents has revealed that this is because opioids – chemicals that have a morphine-like effect on the body – are called into operation during the conditioning and they end up blunting the pain. Blocking the opioids not only stops the pain from being lessened, but also intensifies the learning process. © Copyright Reed Business Information Ltd

Keyword: Emotions; Pain & Touch
Link ID: 11652 - Posted: 06.24.2010

By Steve Mitchell Trust forms the foundation of healthy relationships, and now scientists are zeroing in on how the feeling is triggered by chemicals in the brain. A new study shows that the hormone oxytocin may spur us to trust others even after they have betrayed us by suppressing a region of the brain that signals fear. The findings could lead to a better understanding of social phobias and related disorders. Previous research has shown that oxytocin increases our feelings of trust and plays an important role in bonding with others (ScienceNOW, 1 June 2005). But the areas of the brain it acts on to produce that effect have remained a mystery. To get a better handle on how the hormone affects our noggins, Thomas Baumgartner, a neuroscientist at the University of Zürich, Switzerland, and colleagues monitored the brain activity of 49 men while they engaged in a game involving trust and betrayal. In the game, the men were given money that they could share with another person who might increase the funds through investments and split the profits or betray them and keep all the money. When volunteers got a whiff of oxytocin via a nasal spray, their trust did not diminish even when the second player kept the money to himself half the time. In contrast, men who received a placebo spray reduced the amount of money they forked over. © 2008 American Association for the Advancement of Science.

Keyword: Emotions; Hormones & Behavior
Link ID: 11651 - Posted: 06.24.2010

Barbara Axt It may not be able to catch mice yet, but software developed in the US can perceive moving images in much the same way a cat's brain does. The researchers hope the work will one day lead to implants that make it possible for people to see without an optic nerve. Researchers at the Smith-Kettlewell Eye Research Institute, San Francisco, were motivated by the fact that, until now, models of the way brains respond to visual input used simple images like dots, bars and grids. They are typically unable to cope with the complex scenes a human would usually see. To try and develop a more sophisticated model, the team recorded the responses of 49 individual neurons in a part of a cat's brain called the lateral geniculate nucleus (LGN). The LGN receives and processes visual information from the retina, via the optic nerve, before sending it on to the cerebral cortex. Using a mixture of simple stimuli, like dots and bars, and building up to more complex moving artificial scenes, the team tried to work out the basics of the LGN's response to visual features. Call in the catcamThe data made it possible to build a software model of the LGN that can approximate how the neurons would respond to real scenes. The model was tested against scenes recorded from a "catcam" camera attached to a cat's head. © Copyright Reed Business Information Ltd

Keyword: Vision; Robotics
Link ID: 11650 - Posted: 06.24.2010

HealthDay News) -- Sleep deprivation can affect your ability to make sense of what you see, a study by neuroscience researchers at the Duke-NUS Graduate Medical School in Singapore shows. Using MRI to measure blood flow in the brains of volunteers, the researchers found that even after sleep deprivation, participants had periods of near-normal brain function in which they were able to complete tasks quickly. However, periods of slow response and severe declines in visual processing were mixed in with these periods of normalcy. "Interestingly, the team found that a sleep-deprived brain can normally process simple visuals, like flashing checkerboards. But the 'higher visual areas' -- those that are responsible for making sense of what we see -- didn't function well. Herein lies the peril of sleep deprivation," study author Dr. Michael Chee, a professor of the neurobehavioral disorders program at Duke-NUS, said in a prepared statement. During these slow visual responses, the volunteers showed significant reductions in their higher visual cortex activity. At the same time, their frontal and parietal "control regions" were less able to make their usual connections. The mixture of sleep deprivation-related cognitive lapses and near normalcy demonstrate the competing effects of trying to remain awake while the brain is trying to power down for sleep, when it ordinarily becomes less responsive to sensory stimuli, Chee said. © 2008 U.S. News & World Report, L.P.

Keyword: Sleep; Vision
Link ID: 11649 - Posted: 06.24.2010