Derek Boogaard's relatives and fans shed tears Sunday as they remembered the former NHL tough guy as a "teddy bear" who was as generous and kind as he was burly and tough, a somber end to a weekend during which his distraught family agreed to donate his brain to medical researchers. The 28-year-old Boogaard was found dead in his Minneapolis apartment Friday, five months after he sustained a season-ending concussion with the New York Rangers. Boogaard's agent and a spokeswoman for the Boston University School of Medicine confirmed Sunday that his brain will be examined for signs of a degenerative disease often found in athletes who sustain repeated hits to the head. "It's an amazing thing he did and his family did. Hopefully, that'll bring some information," agent Ron Salcer said. "We don't know exactly the impact that the concussions might have played." Salcer spent three days with Boogaard in Los Angeles earlier in the week. Salcer remarked about his client's brightened demeanor, after suffering through a winter of not being able to play or even be active while his head healed. "He seemed very good, and that's what makes it more painful," Salcer said. "He was really starting to feel better about everything. He was in great shape." Minneapolis police said there were no outward signs of trauma, but results of an autopsy are expected to take several weeks. There is no known concussion connection to his death, but at Boogaard's wish his family signed papers to donate his brain to the BU Center for the Study of Traumatic Encephalopathy. The donation was first reported by the Star Tribune of Minneapolis. © CBC 2011

Keyword: Brain Injury/Concussion
Link ID: 15333 - Posted: 05.16.2011

By PAMELA PAUL NOBODY would deny that being ostracized on the playground, mocked in a sales meeting or broken up with over Twitter feels bad. But the sting of social rejection may be more like the ouch! of physical pain than previously understood. New research suggests that the same areas in the brain that signify physical pain are activated at moments of intense social loss. “When we sat around and thought about the most difficult emotional experiences, we all agreed that it doesn’t get any worse than social rejection,” said the study’s lead author, Ethan F. Kross, an assistant professor of psychology at the University of Michigan. The image of a bunch of social scientists inflicting pain on laboratory volunteers seems creepily Mengelian, but in this case the experiments involved were markedly less cruel. First off, the subjects weren’t socially rejected by the laboratory technicians — each of the 40 volunteers was recruited specifically because he or she felt intensely rejected as a result of a recent (unwanted) breakup. Once in the lab, participants were hooked up to functional M.R.I. scanners, which measure brain activity. They were then asked to look at photos of their former lovers and brood over a specific rejection experience involving that person. (Sob.) Later, they were asked to look at a photograph of a friend and to think about a recent positive experience they had with that person. On to more fun! Next was the physical pain component, also in two parts. First, participants experienced noxious thermal stimulation on their left forearms (the “hot trial”), simulating the experience of spilling hot coffee on themselves. Then, they underwent a second, nonnoxious thermal stimulation (the “warm trial”). Technicians monitored their brain activity to see which areas lighted up. © 2011 The New York Times Company

Keyword: Emotions; Pain & Touch
Link ID: 15332 - Posted: 05.16.2011

By Adam Summers If you’ve ever chased a cat that’s trying to avoid a bath, you have every right to conclude that, for our size, we humans are pretty poor runners. But chasing a cat is sprinting. Where we excel is endurance running. Moreover, we run long distances at fast speeds: many joggers do a mile in seven-and-a-half minutes, and top male marathoners can string five-minute miles together for more than two hours. A quadruped of similar weight, about 150 pounds, prefers to run a mile at a trot, which takes nine-and-a-half minutes, and would have to break into a gallop to keep pace with a good recreational jogger. That same recreational jogger could keep up with the preferred trotting speed of a thousand-pound horse. Good endurance runners are rare among animals. Although humans share the ability with some other groups, such as wolves and dogs, hyenas, wildebeest, and horses, we alone among primates can run long distances with ease. But what evidence can support the idea that endurance running by itself gave early humans an evolutionary advantage, and that it wasn’t just “piggybacking” on our ability to walk? Many traits, after all, are useful for both activities; long legs, for instance, and the long stride they enable, are helpful to walking as well as to running. But running and walking are mechanically different gaits. A walking person, aided by gravity, acts as an inverted pendulum: the hip swings over the planted foot. In contrast, a runner bounces along, aided by tendons and ligaments that act as springs, which alternately store and release energy. © 2008–2011 Natural History Magazine, Inc

Keyword: Evolution
Link ID: 15331 - Posted: 05.16.2011

By ROBERT H. FRANK THE late Amos Tversky, a Stanford psychologist and a founding father of behavioral economics, used to say, “My colleagues, they study artificial intelligence; me, I study natural stupidity.” In recent decades, behavioral economics has been the economics profession’s runaway growth area. Scholars in this field work largely at the intersection of economics and psychology, and much of their attention has focused on systematic biases in people’s judgments and decisions. They point out, for example, that people are particularly inept at predicting how changes in their life circumstances will affect their happiness. Even when the changes are huge — positive or negative — most people adapt much more quickly and completely than they expected. Such prediction errors, behavioral economists argue, often lead to faulty decisions. A celebrated example describes an assistant professor at a distinguished university who agonizes for years about whether he will be promoted. Ultimately, his department turns him down. As anticipated, he’s abjectly miserable — but only for a few months. The next year, he’s settled in a new position at a less selective university, and by all available measures is as happy as he’s ever been. The ostensible lesson is that if this professor had been acquainted with the relevant evidence, he’d have known that it didn’t make sense to fret about his promotion in the first place — that he would have been happier if he hadn’t. But that’s almost surely the wrong lesson, because failing to fret probably would have made him even less likely to get the promotion. And promotions often matter in ways that have little impact on day-to-day levels of happiness. © 2011 The New York Times Company

Keyword: Emotions; Evolution
Link ID: 15330 - Posted: 05.16.2011

By Deborah Kotz, Globe Staff Getting baby to sleep through the night is one of the biggest challenges new parents face. We agonize over the right methods to employ and when to start employing them. Over my husband's objections, I "Ferberized" our first child at less than three months old, letting her cry herself to sleep, after reading "Solve Your Child's Sleep Problems" by Dr. Richard Ferber, a pediatrician at Children's Hospital Boston. It worked like a dream. But my daughter developed some issues with insomnia in early elementary school just around the time my marriage had hit a rough patch. I never made the connection between the two -- since they were both temporary -- but perhaps I should have. An intriguing new study published online yesterday in the journal Child Development finds that marital problems can cause sleep problems in children as young as nine months of age. The researchers initially looked at 357 babies at 9 months of age and found that those whose parents reported high levels of marital instability -- having frequent fights or contemplating divorce -- were more likely to have trouble falling and staying asleep than those whose parents didn't report marriage problems. They were also more likely to have those problems persist at 18 months of age. (Interestingly, the study didn't find that poor-sleeping children triggered marital conflict in their sleep-deprived parents -- which is surprising news to me.) © 2011 NY Times Co

Keyword: Sleep; Development of the Brain
Link ID: 15329 - Posted: 05.14.2011

By LESLEY ALDERMAN WHEN Liz Goldberg, 53, was growing up, she always felt “a little off.” She received good grades and even completed a master’s degree in health administration, but it was always a struggle. In school, she would procrastinate and then pull desperate all-nighters to study for an exam. She’d become hyperfocused on a project and let everything else fall by the wayside. Maintaining relationships was tricky. “I would concentrate intensely on a friend and then move on,” she said. She commuted to college one year simply because she had missed the deadline to apply for housing. “I managed to achieve a lot, but it was difficult,” said Ms. Goldberg, a mother of three who lives near Philadelphia. “I sensed something was wrong, but others would always talk me out of it.” Finally, in her late 40s, Ms. Goldberg was given a diagnosis of attention deficit hyperactivity disorder, a condition caused by signaling problems in the brain. The primary symptoms are impulsiveness, inattention, restlessness and poor self-regulation. Children with the condition tend to be hyperactive, but adults who have it often just seem distracted and disorganized. Undiagnosed, A.D.H.D. can wreak havoc on relationships, finances and one’s self-esteem. Adults with the disorder are twice as likely as those without it to be divorced, for instance, and four times as likely to have car accidents. It’s no surprise that they also tend to have poor credit ratings. © 2011 The New York Times Company

Keyword: ADHD
Link ID: 15328 - Posted: 05.14.2011

By NICHOLAS BAKALAR Ants called Pseudomyrmex triplarinus live inside the leaves and trunk of Triplaris americana trees, where they take shelter and eat sugars, fats and proteins supplied by the tree. In return, they bite animals that try to eat the trees’ leaves, and they prune away plants that grow near them. Now researchers have figured out one way in which they can distinguish a foreign plant from their own. A study showed that Pseudomyrmex triplarinus ants were able to recognize extract from different types of trees independent of the shape or texture of the material that carried it. The scientists, working in Peru, found that the ants consistently pruned foreign seedlings that sprouted near their tree. They also removed 80 to 100 percent of foreign leaves experimentally pinned to the trunk, compared with only 10 to 30 percent of T. americana leaves. Then the investigators treated identical strips of filter paper with leaf wax extracts from T. americana; with extracts from T. poeppigiana, a closely related species; or with plain solvents as a control. The ants attacked the control strips more often than the T. poeppigiana, and the T. poeppigiana more often than the T. americana strips. This suggests the ants could, to a significant degree, recognize the extract independent of the shape or texture of the material that carried it. © 2011 The New York Times Company

Keyword: Chemical Senses (Smell & Taste); Evolution
Link ID: 15327 - Posted: 05.14.2011

Amber Dance Consciousness is not all or nothing: some people languish for months or years in a middling state. A person could have some sense of and interaction with the world, or could be completely unconscious of their surroundings. But today in Science, researchers report a test that can distinguish between states of consciousness using a simple electroencephalogram (EEG) and some mathematics1. The test suggests that the key difference between minimally conscious and totally unconscious non-coma states is communication between the frontal cortex — the planning, thinking part of the brain — and the temporal cortex, where sounds and words are processed. The researchers say that the technique could help doctors to make accurate diagnoses about consciousness, and better predict how a patient will recover. Having this extra information might help families to make difficult decisions about a loved one's care or end of life in cases such as that of Terri Schiavo, who was removed from life support in 2005 after several years in a vegetative state. A person in a vegetative state will open their eyes spontaneously and make reflexive movements, but has no cognitive function and likely does not feel pain. Recovery is possible, but the chances of improvement are greatly diminished after a year. Someone in a minimally conscious state, by contrast, has intentional, non-reflexive but inconsistent responses to stimuli. They might speak a few words or track their image in a mirror, and they feel pain. The category has only been an official diagnosis since 2002, so the likelihood of recovery is not established, but again it decreases after one year in the state. © 2011 Nature Publishing Group,

Keyword: Attention
Link ID: 15326 - Posted: 05.14.2011

By Daniel Strain Talk between the brain’s decision-making center, or frontal cortex, and other brain regions might distinguish aware individuals from those stripped of conscious thought. Identifying such signaling malfunctions could speed the diagnosis of vegetative states and give scientists insight into such devastating disorders, an international team of researchers reports May 12 in Science. Today, diagnosing a vegetative brain is an uncertain enterprise, says John Whyte, director of the Moss Rehabilitation Research Institute in Elkins Park, Pa. Patients classified as vegetative can’t act in any purposeful way under any observable circumstances. Patients deemed minimally conscious, however, show some capacity to understand and interact with the world — for instance, by moving a finger on command. Distinguishing between the two can take weeks of behavioral testing, and misdiagnoses are common. With better diagnostics in mind, Mélanie Boly of the University of Liège in Belgium and colleagues monitored the brain responses of 22 healthy volunteers, 13 patients previously diagnosed as minimally conscious and eight diagnosed as vegetative to fluctuating blips of sound. Minimally conscious and healthy subjects responded to a surprising change in the pitch of the sounds with bursts of brain activity about 170 milliseconds long. Responses from vegetative patients lasted less than 100 milliseconds. Using statistical tools, the researchers filtered through a number of possible causes of such shortened brain activity before determining the most likely culprit. The team concluded that damage to communication paths within the brain, not just to individual regions like the frontal cortex, seemed to typify the vegetative state. © Society for Science & the Public 2000 - 2011

Keyword: Attention
Link ID: 15325 - Posted: 05.14.2011

By John Roach "Blueberry!" I tell my 15-month-old son as I hand him one, hoping that he makes the connection between the piece of fruit and its name as I daydream about the glorious day when he says, "Please, Dad, can I have another blueberry?" For now, he points at the bowl full of tasty morsels and babbles something incomprehensible. His pediatrician, family and friends all assure me that he's on the right track. Before I know it, he'll be rattling off the request for another blueberry and much, much more. This pointing and babbling is all a part of the language learning process, they say, even though the process itself remains largely a mystery. One prominent, though controversial, hypothesis is that some knowledge of grammar is hardwired into our brains. "There's some knowledge that the learner has that actually makes this process easier," Jennifer Culbertson, a postdoctoral fellow at the University of Rochester, explained to me today. This hypothesis was originally proposed 50 years ago by philosopher and linguist Noam Chomsky at the Massachusetts Institute of Technology. Culbertson recently confirmed it with an experiment featuring a virtual green blob for a teacher named Glermi who speaks a nonsensical language called Verblog. © 2011 msnbc.com

Keyword: Language; Development of the Brain
Link ID: 15324 - Posted: 05.14.2011

By Daniel Bates The key to being intelligent could be a thick ‘insulation’ on the brain’s wires, scientists have discovered. A fatty layer covering the neural wires helps brain signals travel faster and makes the brain work more quickly. Just as a thick coating on an electrical wire stops current leaking out, a good layer of insulation helps the brain’s ‘circuits’ function more efficiently. A magnified portion of the brain created using diffusion imaging. The bright red areas show the thick fibre tract - the corpus callosum - which transfers information between the left and right sides of the brain A magnified portion of the brain created using diffusion imaging. The bright red areas show the thick fibre tract - the corpus callosum - which transfers information between the left and right sides of the brain The research is among the first to link ‘neural architecture’ to the health of individuals. It also suggests that this characteristic is something we are born with, indicating that intelligence is something we inherit. The researchers from the University of California in Los Angeles studied images of brain scans from 92 sets of identical twins to determine the amount of myelin - a fatty layer - around the brain’s neural wires. © Associated Newspapers Ltd

Keyword: Intelligence; Brain imaging
Link ID: 15323 - Posted: 05.14.2011

By CELIA WATSON SEUPEL “Why are we in this tunnel?” my mother asks sharply from the back seat. Jolted from my reverie, I turn down the audiobook, lean forward over the steering wheel and look out into the night. The sky is black, brushed with a few pale stars but no moon. Darkness presses on the car windows as we barrel down the highway. The only lights are headlights from oncoming cars, plus the dim glow of the dashboard illuminating my son’s knees. He’s asleep beside me, out cold. Overtired, I repress a wave of irritated giggles. “We’re not in a tunnel, Ma,” I say. I encouraged Mom to stay home while I took this exhausting, daylong round trip to pick up my son, Spencer, from college, but she insisted on coming. Mom loves riding in the car, and she’s always bragging to her friends about what a great driver I am. I hear her rustle as she turns in the back seat, looking out the windows. I know she doesn’t believe me. “Well, where are we?” she demands. “We’re in the car,” I say. I grab my emergency Hershey’s chocolate almond bar and rip off the wrapper with my teeth. “I can see we’re in the car, Celia,” she says scornfully. I picture my mother sitting bolt upright behind me, straightening her glasses on her nose, clutching her puffy winter coat more tightly. © 2011 The New York Times Company

Keyword: Alzheimers
Link ID: 15322 - Posted: 05.12.2011

Analysis by Nic Halverson "A mental breakdown in the relation between thought, emotion and behavior, leading to faulty perception, inappropriate actions and feelings, withdrawal from reality and personal relationships into fantasy and delusion, and a sense of mental fragmentation." Sound like what your brain feels like after an Angry Birds marathon or an 8-hour binge of searching for the end of the Internet? Maybe. But this is actually the definition of schizophrenia. Ubiquitous computing might send schizophrenic ripples through your frontal lobes whenever you log on, but researchers at the University of Texas at Austin are taking that a step further by actually using computers to simulate the mental disorder. One theory about the cause of schizophrenia suggests that an afflicted brain remembers too many irrelevant things, due to an excessive release of dopamine. Overwhelmed by vast caches of facts, thoughts and memories all junk-piled in their heads, schizophrenics start processing them into delusional conclusions not based in reality. Uli Grasemann, a graduate student in the University of Texas' Department of Computer Science, used a synthetic neural network to simulate these delusions, often called hyperlearning hypothesis. Designed by his adviser, professor Risto Miikkulainen, Graseman used the network, dubbed DISCERN,to mimic the effects that different neurological dysfunction have on human language function. © 2011 Discovery Communications, LLC

Keyword: Schizophrenia; Robotics
Link ID: 15321 - Posted: 05.12.2011

By Tina Hesman Saey Women with lower levels of the hormone oxytocin in their blood during late stages of pregnancy are more likely to develop postpartum depression, new research suggests. The finding, published online May 11 in Neuropsychopharmacology, may be a first step toward identifying pregnant women who are at risk of becoming depressed after the birth of their babies. While the link is intriguing, “there’s a lot of steps to go between a correlation and using it as a biomarker,” says Ziad Nahas, a psychiatrist and mood disorders researcher at the Medical University of South Carolina in Charleston who was not involved in the study. Oxytocin helps stimulate contractions and lactation in pregnant women. The hormone has also been implicated in facilitating emotional bonds between people, including mothers and children. Some earlier studies have linked oxytocin to depression, but not to postpartum depression. Of 73 women who volunteered blood samples for the study, 14 later developed postpartum depression. Oxytocin concentrations in the women’s blood varied widely, from 14.4 to 245.7 picograms per milliliter. Although the researchers found a great deal of overlap in oxytocin levels between the two groups, women who got depressed tended to have lower levels. The link is too preliminary for doctors to use oxytocin levels to predict which patients are likely to develop postpartum depression, says Gunther Meinlschmidt, a psychobiologist at the University of Basel in Switzerland who led the study. Even if a woman has low levels of the hormone, there’s no guarantee she will get depressed. “This is not a yes, no, one-or-zero factor. It’s one of several factors” that determine a woman’s risk for the condition, he says. It’s also unclear whether treating women with oxytocin would have any effect on depression. © Society for Science & the Public 2000 - 2011

Keyword: Depression; Hormones & Behavior
Link ID: 15320 - Posted: 05.12.2011

by Andy Coghlan A STRANGE property marks out the brain cells of people with Alzheimer's: they have a glut of cells with more than the standard two sets of chromosomes. Furthermore, these turn out to be the cells most likely to perish in the late stages of the disease. The twin discoveries could drive research into one of the modern world's most devastating neurological conditions in an entirely new direction. No one can agree on the cause of Alzheimer's disease. While much of the focus has been on the plaques that clog up the brains of sufferers, treatments that clear these plaques have no effect on the symptoms of the disease. And treatments that block the beta-amyloid proteins that make up the plaques, or the tau proteins that develop within neurons. Perhaps that's because the disease is triggered by something completely different. Last year, Thomas Arendt's team at the University of Leipzig in Germany examined tissue taken from healthy brains and from the brains of those who had Alzheimer's at the time of death, or who showed signs of being about to develop the disease. They found that about 10 per cent of neurons in the brains of healthy people contained more than two sets of chromosomes, a condition known as hyperploidy. The finding is astonishing because all cells in the human body - other than sperm and eggs - are supposed to contain just two sets of chromosomes. More importantly, in the period just before Alzheimer's develops and in the early stages of the disease, Arendt and colleagues found that hyperploid cells double in number. Then, in the final stage of Alzheimer's, most of these unusual cells disappear altogether - of the brain cells lost, 90 per cent are hyperploid (see diagram) (American Journal of Pathology, DOI: 10.2353/ajpath.2010.090955). © Copyright Reed Business Information Ltd.

Keyword: Alzheimers; Genes & Behavior
Link ID: 15319 - Posted: 05.12.2011

by Elizabeth Norton Lasley The waters of the North Sea are among the murkiest on the planet, so dark and silty that a seal sometimes can’t see its own whiskers. Even so, the harbor seals there can hunt and catch fish. Marine biologists have known for several years that a seal relies on its whiskers to follow the wake a fish leaves behind. But according to a new study, whiskers supply detailed information that the seal may use to decide which fish are most worthwhile to hunt. To find out just what a seal’s whiskers can tell it, biologist Wolf Hanke of the University of Rostock in Germany and colleagues enlisted the aid of a harbor seal named Henry, a veteran research participant who has been at the University of Rostock’s Marine Science Center since 2008. “When he starts working, he can’t be stopped,” Hanke says. The researchers presented Henry with different types of wakes using a test box of still water about 2 meters wide that sat at the bottom of a pool about 1 meter deep. Above the box hung a motorized arm with a series of paddles of different shapes and widths. While each paddle moved through the water, Henry waited outside the test box wearing headphones to block out the motor noise. He also wore a blindfold to ensure that he’d be using only his whiskers. Once the paddle had come to rest, Henry left his station and dunked his body halfway into the test box. With the water swirling around his whiskers, Henry made his decision, then left the box and pressed one of two buttons with his nose, indicating whether the wake had been made by one of three paddle sizes he’d already been trained to recognize or by an unfamiliar one. © 2010 American Association for the Advancement of Science.

Keyword: Pain & Touch
Link ID: 15318 - Posted: 05.12.2011

by Kayt Sukel Our intrepid reporter performs an intimate act in an fMRI scanner to explore the pathways of pleasure and pain WITH a click and a whirr, I am pulled into the scanner. My head is strapped down and I have been draped with a blanket so that I may touch my nether regions - my clitoris in particular - with a certain degree of modesty. I am here neither for a medical procedure nor an adult movie. Rather, I am about to stimulate myself to orgasm while an fMRI scanner tracks the blood flow in my brain. My actions are helping Barry Komisaruk at Rutgers University in Newark, New Jersey, and colleagues to tease apart the mechanisms underlying sexual arousal. In doing so, not only have they discovered that there is more than one route to orgasm, but they may also have revealed a novel type of consciousness - an understanding of which could lead to new treatments for pain (see Top-down pain relief). Despite orgasm being a near-universal human phenomenon, we still don't know all that much about it. "The amount of speculation versus actual data on both the function and value of orgasm is remarkable," says Julia Heiman, director of the Kinsey Institute for Research in Sex, Gender and Reproduction in Bloomington, Indiana. It is estimated that one in four women in the US has had difficulty achieving orgasm in the past year, while between 5 and 10 per cent of women are anorgasmic - unable to achieve orgasm at all. But without precise data to explain what happens during this experience, there are few treatment options available for women who might want help. © Copyright Reed Business Information Ltd.

Keyword: Sexual Behavior; Attention
Link ID: 15317 - Posted: 05.12.2011

Ewen Callaway The first humans to reach Europe may have found it a ghost world. Carbon-dated Neanderthal remains from the foothills of the Caucasus Mountains suggest that the archaic species had died out before modern humans arrived. The remains are almost 10,000 years older than expected. They come from just one cave in western Russia, called Mezmaiskaya, but bones at other Neanderthal sites farther west could also turn out to be more ancient than previously thought, thanks to a precise carbon-dating technique, says Thomas Higham, a palaeoanthropologist at the University of Oxford, UK, and a co-author of a study published this week in the Proceedings of the National Academy of Sciences1. The implication, says Higham's team, is that Neanderthals and humans might never have met in Europe. However, the Neanderthal genome, decoded last year2, hints that the ancestors of all humans, except those from Africa, interbred with Neanderthals somewhere. Perhaps humans departing Africa encountered resident Neanderthals in the Middle East. "DNA results show that there was admixture probably at some stage in our human ancestry, but it more than likely happened quite a long time before humans arrived in Europe," says Ron Pinhasi, an archaeologist at University College Cork in Ireland, who is lead author of the latest study. "I don't believe there were regions where Neanderthals were living next to modern humans. I just don't find it very feasible," he adds. © 2011 Nature Publishing Group,

Keyword: Evolution
Link ID: 15316 - Posted: 05.10.2011

By GARDINER HARRIS Nearly one in seven elderly nursing home residents, nearly all of them with dementia, are given powerful atypical antipsychotic drugs even though the medicines increase the risks of death and are not approved for such treatments, a government audit found. More than half of the antipsychotics paid for by the federal Medicare program in the first half of 2007 were “erroneous,” the audit found, costing the program $116 million for those six months. “Government, taxpayers, nursing home residents as well as their families and caregivers should be outraged and seek solutions,” Daniel R. Levinson, inspector general of the Department of Health and Human Services, wrote in announcing the audit results. Mr. Levinson noted that such drugs — which include Risperdal, Zyprexa, Seroquel, Abilify and Geodon — are “potentially lethal” to many of the patients getting them and that some drug manufacturers illegally marketed their medicines for these uses “putting profits before safety.” The audit is an unusual assessment by the government of whether doctors are treating Medicare patients appropriately in nursing homes. Mr. Levinson suggested that the government should collect information on the diagnoses given Medicare patients so that the government can assess whether the drugs prescribed to them are appropriate. © 2011 The New York Times Company

Keyword: Alzheimers; Schizophrenia
Link ID: 15315 - Posted: 05.10.2011

By PERRI KLASS, M.D. The mother had brought in a note from her son’s elementary school teacher: Dear doctor, I think this child needs to be tested for attention deficit disorder. “She’s worried about how he can’t sit still in school and do his work,” the mother said. “He’s always getting into trouble.” But then she brightened. “But he can’t have attention deficit, I know that.” Why? Her son could sit for hours concentrating on video games, it turned out, so she was certain there was nothing wrong with his attention span. It’s an assertion I’ve heard many times when a child has attention problems. Sometimes parents make the same point about television: My child can sit and watch for hours — he can’t have A.D.H.D. In fact, a child’s ability to stay focused on a screen, though not anywhere else, is actually characteristic of attention deficit hyperactivity disorder. There are complex behavioral and neurological connections linking screens and attention, and many experts believe that these children do spend more time playing video games and watching television than their peers. But is a child’s fascination with the screen a cause or an effect of attention problems — or both? It’s a complicated question that researchers are still struggling to tease out. © 2011 The New York Times Company

Keyword: ADHD; Attention
Link ID: 15314 - Posted: 05.10.2011