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by Linda Geddes They say the early bird catches the worm, but night owls may be missing far more than just a tasty snack. Researchers have discovered the first physical evidence of structural brain differences that distinguish early risers from people who like to stay up late. The differences might help to explain why night owls seem to be at greater risk of depression. Around 10 per cent of people qualify as morning people or larks, and a further 20 per cent are night owls – with the rest of us falling somewhere in between. Your lark or night owl status is called your chronotype. Previous studies have suggested that night owls experience worse sleep, more tiredness during the day and consume greater amounts of tobacco and alcohol. This has prompted some to suggest that they are suffering from a form of chronic jet lag. To investigate further, Jessica Rosenberg at RWTH Aachen University in Germany and colleagues used diffusion tensor imaging to scan the brains of 16 larks, 23 night owls and 20 intermediate chronotypes. They found a reduction in the integrity of night owls' white matter – brain tissue largely comprised of fatty insulating material that speeds up the transmission of nerve signals – in areas associated with depression. "We think this could be caused by the fact that late chronotypes suffer from this permanent jet lag," says Rosenberg, although she cautions that further studies are needed to confirm cause and effect. © Copyright Reed Business Information Ltd.
Keyword: Biological Rhythms; Depression
Link ID: 18731 - Posted: 10.01.2013
By Julianne Wyrick Some people are drawn to the thick smell of bacon, sizzling and crackling in the skillet on a Saturday morning. For others, it’s the aroma of freshly baked cookies on a Friday night or the smell of McDonald’s fries creeping in through the car window. At this time of year, I find the scent of freshly baked pumpkin muffins irresistible. Of course, I’d like to think I’m not a slave to my nose, at least not when I’m nice and full from dinner. If I were a fruit fly, my outlook might not be so good. Already-fed fruit fly larvae exposed to certain food-related odors ate more food than larvae that didn’t experience the smells, according to research published by scientists at the University of Georgia last spring. “They’re not hungry, but they will get an extra kick in terms of appetite, so they will eat, for example, 30 percent extra,” said Ping Shen, lead author on the study. The scents, which included the sweet odor of bananas or the sharper smell of balsamic vinegar, served as “cues” or triggers that the flies associated with food. The triggers motivated the fly larvae to eat, even when they’d already had dinner. That doesn’t bode so well for flies trying to watch their weight. For the fly to feel this urge to eat, the smell has to be transported from sensory receptors in the nose to the part of the brain that regulates appetite—the brain’s “feeding center”—via a series of neurons. Part of this signal transfer involves dopamine, a neurotransmitter associated with behavior motivated by a cue or hint of something to come, like smells associated with food. © 2013 Scientific American
Keyword: Obesity; Chemical Senses (Smell & Taste)
Link ID: 18730 - Posted: 10.01.2013
By NICHOLAS BAKALAR Black and Hispanic children who go to an emergency room with stomach pain are less likely than white children to receive pain medication, a new study reports, and more likely to spend long hours in the emergency room. The analysis, published in the October issue of Pediatrics, examined the records of 2,298 emergency room visits by people under 21, a nationally representative sample from a large survey conducted by the Centers for Disease Control and Prevention. About 53 percent were white, 24 percent non-Hispanic black, 21 percent Hispanic, and the rest from other ethnic or racial groups. Over all, 27.1 percent of white children with severe pain received analgesics, but only 15.8 percent of blacks, 18.9 percent of Hispanics and 7.1 percent of children of other races did. Black children were about 68 percent more likely than white children to spend longer than six hours in the emergency room, although there were no statistically significant differences among races in results for any diagnostic test. “This data set will not answer the question of why,” said the lead author, Dr. Tiffani J. Johnson, an instructor at the University of Pennsylvania School of Medicine. “It could be that white parents are more likely to ask for pain meds, or that minority patients are likely to get care in E.R.’s that have longer wait times. And it could be racial bias.” Copyright 2013 The New York Times Company
Keyword: Pain & Touch; Emotions
Link ID: 18729 - Posted: 10.01.2013
By Kendall Powell, Although my grandmother received a diagnosis of Alzheimer’s disease in her 80s, my family was never sure that’s what she had. She certainly suffered from dementia: She was able to recall childhood memories but couldn’t remember what she had had for lunch. But dementia and Alzheimer’s are not synonymous. Back then, the only way to look for the telltale Alzheimer’s plaques — deposits of the protein fragment beta amyloid that accumulate in the spaces between nerve cells — was through an autopsy, which we didn’t do. Over the past 15 years, researchers have developed a greater understanding of how the disease works. We now have more accurate ways of diagnosing Alzheimer’s and are moving closer to developing drugs to directly attack the disease. Much of this work is still in the early stages, but experts are growing more hopeful about dealing with the debilitating disease, which currently has no cure. Now, for example, we no longer have to rely on autopsies to confirm the existence of Alzheimer’s plaques. In a major advance last year, the Food and Drug Administration approved a method that uses a radioactive dye, known commercially as Amyvid, to light up amyloid plaques in a PET scan. The FDA approved Amyvid to rule out Alzheimer’s when the scan is negative and to confirm the presence of plaques when positive, but that does not necessarily indicate the disease is present. However, some doctors are using the scans to confirm the disease, which experts say is misdiagnosed up to a quarter of the time. Paul Aisen, director of the Alzheimer’s Disease Cooperative Study at University of California at San Diego, calls Amyvid an “enormous advance” because a positive scan, combined with his clinical diagnosis, means he can tell patients and their families the disease is “present, not probable.” © 1996-2013 The Washington Post
Keyword: Alzheimers
Link ID: 18728 - Posted: 10.01.2013
Mid-life stress may increase a woman's risk of developing dementia, according to researchers. In a study of 800 Swedish women, those who had to cope with events such as divorce or bereavement were more likely to get Alzheimer's decades later. The more stressful events there were, the higher the dementia risk became, BMJ Open reports. The study authors say stress hormones may be to blame, triggering harmful alterations in the brain. Stress hormones can cause a number of changes in the body and affect things such as blood pressure and blood sugar control. And they can remain at high levels many years after experiencing a traumatic event, Dr Lena Johansson and colleagues explain. But they say more work is needed to confirm their findings and ascertain whether the same stress and dementia link might also occur in men. In the study, the women underwent a battery of tests and examinations when they were in either their late 30s, mid-40s or 50s, and then again at regular intervals over the next four decades. At the start of the study, one in four women said they had experienced at least one stressful event, such as widowhood or unemployment. BBC © 2013
Keyword: Stress; Alzheimers
Link ID: 18727 - Posted: 10.01.2013
By Larry Greenemeier The leaves of the herb kratom (Mitragyna speciosa), a native of Southeast Asia in the coffee family, are used to relieve pain and improve mood as an opiate substitute and stimulant. The herb is also combined with cough syrup to make a popular beverage in Thailand called “4x100.” Because of its psychoactive properties, however, kratom is illegal in Thailand, Australia, Myanmar (Burma) and Malaysia. The U.S. Drug Enforcement Administration lists kratom as a “drug of concern” because of its abuse potential, stating it has no legitimate medical use. The state of Indiana has banned kratom consumption outright. Now, looking to control its population’s growing dependence on methamphetamines, Thailand is attempting to legalize kratom, which it had originally banned 70 years ago. At the same time, researchers are studying kratom’s ability to help wean addicts from much stronger drugs, such as heroin and cocaine. Studies show that a compound found in the plant could even serve as the basis for an alternative to methadone in treating addictions to opioids. The moves are just the latest step in kratom’s strange journey from home-brewed stimulant to illegal painkiller to, possibly, a withdrawal-free treatment for opioid abuse. With kratom’s legal status under review in Thailand and U.S. researchers delving into the substance’s potential to help drug addicts, Scientific American spoke with Edward Boyer, a professor of emergency medicine and director of medical toxicology at the University of Massachusetts Medical School. Boyer has worked with Chris McCurdy, a University of Mississippi professor of medicinal chemistry and pharmacology, and others for the past several years to better understand whether kratom use should be stigmatized or celebrated. © 2013 Scientific American
Keyword: Drug Abuse
Link ID: 18726 - Posted: 10.01.2013
Erika Check Hayden The power of thought alone is not enough to move inanimate objects — unless the object is a robotic leg wired to your brain, that is. A 32-year-old man whose knee and lower leg were amputated in 2009 after a motorcycle accident is apparently the first person with a missing lower limb to control a robotic leg with his mind. A team led by biomedical engineer Levi Hargrove at the Rehabilitation Institute of Chicago in Illinois reported the breakthrough last week in the New England Journal of Medicine1, including a video that shows the man using the bionic leg to walk up stairs and down a ramp, and to kick a football. The major advance is that the man does not have to use a remote-control switch or exaggerated muscle movements to tell the robotic leg to switch between types of movements, and he does not have to reposition the leg with his hands when seated, Hargrove says. “To our knowledge, this is the first time that neural signals have been used to control both a motorized knee and ankle prosthesis,” he says. Scientists had previously shown that paralysed people could move robotic arms using their thoughts and that able-bodied people can walk using robotic legs controlled by their brains (see, for example, go.nature.com/dgtykw). The latest work goes a step further by using muscle signals to amplify messages sent by the brain when the person intends to move. © 2013 Nature Publishing Group
Keyword: Robotics
Link ID: 18725 - Posted: 10.01.2013
Selectively bred strains of laboratory rats that either prefer or avoid alcohol have been a mainstay of alcohol research for decades. So-called alcohol-preferring rats voluntarily consume much greater amounts of alcohol than do non-preferring rats. Scientists at the National Institutes of Health now report that a specific gene plays an important role in the alcohol-consuming tendencies of both types of rats. “This study advances our understanding of the genetics and neurobiology of alcohol consumption in an important animal model of human alcoholism,” says Kenneth R. Warren, Ph.D., acting director of the National Institute on Alcohol Abuse and Alcoholism (NIAAA), part of NIH. As reported online in the Proceedings of the National Academy of Sciences, a diverse team of scientists, led by David Goldman, M.D., chief of NIAAA’s Laboratory of Neurogenetics, used exome sequencing, an approach that comprehensively analyzes the DNA that encodes proteins. They found a severely dysfunctional form of the gene for a brain signaling molecule called metabotropic glutamate receptor 2 (Grm2), known as a stop codon, in alcohol-preferring rats but not in non-preferring rats. The researchers then demonstrated that drugs and genetic changes that block Grm2 increased alcohol consumption in normal rats and mice. “We’ve long known that genes play an important role in alcoholism,” says Dr. Goldman. “However, the genes and genetic variants that cause alcoholism have remained largely unknown. This first discovery of a gene accounting for alcohol preference in a mammalian model illustrates that genomic analysis of a model organism is a powerful approach for a complex disease such as alcoholism.”
Keyword: Drug Abuse; Genes & Behavior
Link ID: 18724 - Posted: 10.01.2013
By Tanya Lewis, Look closely at the FedEx logo and you'll notice the space between the "E" and the "x" creates the outline of an arrow. Now, a new study reveals the part of the brain that creates such invisible shapes. The FedEx arrow is just one example of a common optical illusion, whereby the brain "sees" shapes and surfaces within a fragmented background, although they don't exist. Scientists studied the effect in monkeys, finding a group of neurons in part of the visual cortex that fire when the animals viewed an illusion pattern. Besides monkeys, studies have shown that a host of other animals experience shape illusions, including cats, owls, goldfish and honeybees. Scientists think the mental quirk might have evolved to help animals spot predators or prey in the bushes. "Basically, the brain is acting like a detective," study leader Alexander Maier, a psychologist at Vanderbilt University in Nashville, Tenn., said in a statement. "It is responding to cues in the environment and making its best guesses about how they fit together. In the case of these illusions, however, it comes to an incorrect conclusion." The visual cortex, a part of the brain at the back of the head, processes visual information in mammals. Scientists often divide the visual cortex into five regions labeled V1 through V5. Visual signals from the eyes go to the primary visual cortex, V1, which detects their orientation, color and spatial arrangement. The brain splits that information into two streams, known as the dorsal and ventral streams. Both pathways go to V2, which makes some connections to V3.
Keyword: Vision
Link ID: 18723 - Posted: 10.01.2013
By BRANDON A. GAUDIANO PROVIDENCE, R.I. — PSYCHOTHERAPY is in decline. In the United States, from 1998 to 2007, the number of patients in outpatient mental health facilities receiving psychotherapy alone fell by 34 percent, while the number receiving medication alone increased by 23 percent. This is not necessarily for a lack of interest. A recent analysis of 33 studies found that patients expressed a three-times-greater preference for psychotherapy over medications. As well they should: for patients with the most common conditions, like depression and anxiety, empirically supported psychotherapies — that is, those shown to be safe and effective in randomized controlled trials — are indeed the best treatments of first choice. Medications, because of their potential side effects, should in most cases be considered only if therapy either doesn’t work well or if the patient isn’t willing to try counseling. So what explains the gap between what people might prefer and benefit from, and what they get? The answer is that psychotherapy has an image problem. Primary care physicians, insurers, policy makers, the public and even many therapists are largely unaware of the high level of research support that psychotherapy has. The situation is exacerbated by an assumption of greater scientific rigor in the biologically based practices of the pharmaceutical industries — industries that, not incidentally, also have the money to aggressively market and lobby for those practices. For the sake of patients and the health care system itself, psychotherapy needs to overhaul its image, more aggressively embracing, formalizing and promoting its empirically supported methods. My colleague Ivan W. Miller and I recently surveyed the empirical literature on psychotherapy in a series of papers we edited for the November edition of the journal Clinical Psychology Review. It is clear that a variety of therapies have strong evidentiary support, including cognitive-behavioral, mindfulness, interpersonal, family and even brief psychodynamic therapies (e.g., 20 sessions). © 2013 The New York Times Company
Keyword: Depression
Link ID: 18722 - Posted: 09.30.2013
The Conservative government is launching a $1.3-billion free market in medical marijuana on Tuesday, eventually providing an expected 450,000 Canadians with quality weed. Health Canada is phasing out an older system on Monday that mostly relied on small-scale, homegrown medical marijuana of varying quality, often diverted illegally to the black market. In its place, large indoor marijuana farms certified by the RCMP and health inspectors will produce, package and distribute a range of standardized weed, all of it sold for whatever price the market will bear. The first sales are expected in the next few weeks, delivered directly by secure courier. "We're fairly confident that we'll have a healthy commercial industry in time," Sophie Galarneau, a senior official with the department, said in an interview. "It's a whole other ball game." The sanctioned birth of large-scale, free-market marijuana production comes as the Conservatives pillory Liberal Leader Justin Trudeau's campaign to legalize recreational marijuana. Health Canada is placing no limits on the number of these new capital-intensive facilities, which will have mandatory vaults and security systems. Private-dwelling production will be banned. Imports from places such as the Netherlands will be allowed. Already 156 firms have applied for lucrative producer and distributor status since June, with the first two receiving licences just last week. © CBC 2013
Keyword: Drug Abuse; Pain & Touch
Link ID: 18721 - Posted: 09.30.2013
By WILLIAM J. BROAD SCIENCE has looked into some strange things over the centuries — reports of gargantuan sea monsters, purported images of Jesus, sightings of alien spaceships and so on. When I first heard of spontaneous orgasm, while researching a book on yoga, including its libidinal cousin, tantra, I figured it was more allegory than reality and in any event would prove beyond the reach of even the boldest investigators. Well, I was wrong. It turns out science has tiptoed around the subject for more than a century and of late has made considerable progress in determining not only the neurophysiological basis of the phenomenon but also its prevalence. Men are mentioned occasionally. But sex researchers have found that the novel type of autoerotism shows up mainly in women. Ground zero for the research is Rutgers University, where scientists have repeatedly had female volunteers put their heads into giant machines and focus their attention on erotic fantasies — the scans reveal that the pleasure centers of their brains light up in ways indistinguishable from everyday orgasms. The lab atmosphere is no-nonsense, with plenty of lights and white coats and computer monitors. Subjects often thrash about so forcefully that obtaining clear images of their brains can be difficult. “Head movement is a huge issue,” Nan Wise, a doctoral candidate at Rutgers who helps run the project, said in an interview. “It’s hard to get a decent signal.” She said a volunteer’s moving her head more than two millimeters — less than a 10th of an inch — can make for a bad day in the lab. It is easy to dismiss this as a new kind of narcissism in search of scientific respectability, a kinky pleasure coming out of the shadows. Many YouTube videos now purport to show people using controlled breathing and erotic introspection to achieve what they describe as “thinking off” and “energy orgasms.” © 2013 The New York Times Company
Keyword: Sexual Behavior; Brain imaging
Link ID: 18720 - Posted: 09.30.2013
Neuroscience students at the University of Lethbridge are stepping into a maze of memory functions, hoping to help people with dementia or brain injuries through new research that tests people’s memory in the field. That is, the field outside Markin Hall on the U of L’s campus, where neuroscience PhD student Erin Zelinski has set up a life-sized version of a navigation experiment that until now has only been done locally with rats. In it, participants walk around the field until they reach an invisible target spot. When they do, they’ll hear a whistle letting them know they’re in the right place. Then, while researchers time their progress, following their every move with GPS and an overhead remote-controlled camera, participants must find their way back to the same spot two days later. The idea is to first study the brain functions of people without memory impairments so that researchers can later compare that data to a future study of people with memory loss. “We’re trying to describe what normal performance on this test looks like, so if you take a person who’s healthy and you have them perform the task, what we’ll see is that there will probably be commonalities that are going to emerge,” Zelinski said. “And then if you start to look at people that have memory impairments or a brain injury, when they perform the task there might be some things that are different. The better we are at characterizing it in normal people, the better we’re going to be at identifying where the impairments are in those individuals that are having memory problems.” © 1996-2013 The Lethbridge Herald
Keyword: Learning & Memory
Link ID: 18719 - Posted: 09.30.2013
By DAVID P. BARASH WAR is in the air. Sad to say, there’s nothing new about this. Nor is there anything new about the claim that war has always been with us, and always will be. What is new, it seems, is the degree to which this claim is wrapped in the apparent acquiescence of science, especially the findings of evolutionary biology with respect to a war-prone “human nature.” This year, an article in The National Interest titled “What Our Primate Relatives Say About War” answered the question “Why war?” with “Because we are human.” In recent years, a piece in New Scientist asserted that warfare has “played an integral part in our evolution” and an article in the journal Science claimed that “death in warfare is so common in hunter-gatherer societies that it was an important evolutionary pressure on early Homo sapiens.” The emerging popular consensus about our biological predisposition to warfare is troubling. It is not just scientifically weak; it is also morally unfortunate, as it fosters an unjustifiably limited vision of human potential. Although there is considerable reason to think that at least some of our hominin ancestors engaged in warlike activities, there is also comparable evidence that others did not. While it is plausible that Homo sapiens owed much of its rapid brain evolution to natural selection’s favoring individuals that were smart enough to defeat their human rivals in violent competition, it is also plausible that we became highly intelligent because selection favored those of our ancestors who were especially adroit at communicating and cooperating. Conflict avoidance, reconciliation and cooperative problem solving could also have been altogether “biological” and positively selected for. © 2013 The New York Times Company
Keyword: Aggression; Evolution
Link ID: 18718 - Posted: 09.30.2013
By Laura Sanders By hijacking connections between neurons deep within the brain, scientists forced full mice to keep eating and hungry mice to shun food. By identifying precise groups of cells that cause eating and others that curb it, the results begin to clarify the intricate web of checks and balances in the brain that control feeding. “This is a really important missing piece of the puzzle,” says neuroscientist Seth Blackshaw of Johns Hopkins University in Baltimore. “These are cell types that weren’t even predicted to exist.” A deeper understanding of how the brain orchestrates eating behavior could lead to better treatments for disorders such as anorexia and obesity, he says. Scientists led by Joshua Jennings and Garret Stuber of the University of North Carolina at Chapel Hill genetically tweaked mice so that a small group of neurons would respond to light. When a laser shone into the brain, these cells would either fire or, in a different experiment, stay quiet. These neurons reside in a brain locale called the bed nucleus of the stria terminalis, or BNST. Some of the message-sending arms of these neurons reach into the lateral hypothalamus, a brain region known to play a big role in feeding. When a laser activated these BNST neurons, the mice became ravenous, voraciously eating their food, the researchers report in the Sept. 27 Science. “As soon as you turn it on, they start eating and they don’t stop until you turn it off,” Stuber says. The opposite behavior happened when a laser silenced BNST neurons’ messages to the lateral hypothalamus: The mice would not eat, even when hungry. © Society for Science & the Public 2000 - 2013
Keyword: Obesity
Link ID: 18717 - Posted: 09.28.2013
Heather Saul Stress can make the world around us smell unpleasant, the results of a new study are suggesting. Researchers from the University of Wisconsin-Madison used powerful brain imaging technologies to examine how stress and anxiety "re-wire" the brain. A team of psychologists led by Professor Wen Li discovered that when a person experiences stress, emotion systems and olfactory processing in the brain become linked, making inoffensive smells become unpleasant. Although the emotion and olfactory systems within the brain are usually found next to each other, there is rarely 'crosstalk' between the two. Writing in the Journal of Neuroscience, Prof Li said results from their research will now help to uncover the biological mechanisms at work when a person feels stressed. Using functional MRI scans, the team analysed the brain activity of 12 participants after showing them images designed to induce anxiety as they smelled familiar, neutral odours. The subjects were then asked to rate the different smells before being shown the disturbing image and afterwards. The majority showed a more negative response to odours that they had previously considered neutral. This fuels a 'feedback loop' that heightens distress, and can even lead to clinical issues such as depression. Prof Li explained: "After anxiety induction, neutral smells become clearly negative." “In typical odor processing, it is usually just the olfactory system that gets activated,” says Li. “But when a person becomes anxious, the emotional system becomes part of the olfactory processing stream. © independent.co.uk
Keyword: Stress; Chemical Senses (Smell & Taste)
Link ID: 18716 - Posted: 09.28.2013
If you look at the facts and figures on the mental health charity Mind's website, you'll find that around 1 in 4 people will experience some sort of mental health problem each year. About 10% of these people will see their doctor and be diagnosed as having a mental health problem, and of this group, a small proportion will in turn be referred to specialist psychiatric care. Of these people, precisely none resemble the breathtakingly ignorant costumes that have recently been withdrawn from Tesco and Asda. If you want to know what someone with a mental health issue looks like, just look around you. One of the most common types of mental health issue is anxiety – about 9% of people in Britain meet the criteria for mixed anxiety and depression, for example. We all feel anxious from time to time, and that's not necessarily a bad thing. Isaac Marks and Randy Nesse argued in 1994 that anxiety is an important emotion that has been shaped during the course of human evolution. If we are in a potentially dangerous environment, being anxious increases our awareness of our surroundings and puts us in a state of physiological readiness to deal with any threats. However, when an anxiety response kicks in too often, and in situations where it is not needed, it becomes a debilitating problem. In serious cases, anxiety can make it incredibly hard for the person to function. There's now a wealth of research that is trying to tap into the mechanisms involved in both sub-clinical and clinical forms of anxiety. By understanding what happens when we become anxious, we might be able to get a clearer idea of how and why things go wrong in anxiety disorders. For example, a new study published this week in the Journal of Neuroscience has suggested one potential contributing factor – how smells are processed. © 2013 Guardian News and Media Limited
Keyword: Emotions; Chemical Senses (Smell & Taste)
Link ID: 18715 - Posted: 09.28.2013
By Tina Hesman Saey The sun exerts hegemony over biological rhythms of nearly every organism on Earth. But two studies now show the moon is no slouch. It controls the cadence of at least two different biological clocks: one set by tides and the other by moonlight. The clocks, both discovered in sea creatures, work independently of the circadian clock, which synchronizes daily rhythms with the sun. The studies demonstrate that the moon’s light and its gravitational pull, which creates tides, can affect the behavior of animals. “The moon has an influence, definitely,” says Steven Reppert, a neurobiologist at the University of Massachusetts Medical School in Worcester, who was not involved with either study. “Clearly for these marine organisms, it’s very powerful and important.” Scientists established decades ago that circadian clocks govern people’s daily cycles of such things as hormone levels, blood pressure and body temperature. Nearly every organism, including single-celled creatures, has some version. Circadian clocks are composed of protein gears. In a loop that takes roughly 24 hours, levels of some proteins rise and then fall, while others fall and then rise. Sunlight sets the clocks, but once a clock is set it will keep running, even when scientists keep organisms in constant darkness. © Society for Science & the Public 2000 - 2013
Keyword: Biological Rhythms
Link ID: 18714 - Posted: 09.28.2013
By Roy F. Baumeister It has become fashionable to say that people have no free will. Many scientists cannot imagine how the idea of free will could be reconciled with the laws of physics and chemistry. Brain researchers say that the brain is just a bunch of nerve cells that fire as a direct result of chemical and electrical events, with no room for free will. Others note that people are unaware of some causes of their behavior, such as unconscious cues or genetic predispositions, and extrapolate to suggest that all behavior may be caused that way, so that conscious choosing is an illusion. Scientists take delight in (and advance their careers by) claiming to have disproved conventional wisdom, and so bashing free will is appealing. But their statements against free will can be misleading and are sometimes downright mistaken, as several thoughtful critics have pointed out. Arguments about free will are mostly semantic arguments about definitions. Most experts who deny free will are arguing against peculiar, unscientific versions of the idea, such as that “free will” means that causality is not involved. As my longtime friend and colleague John Bargh put it once in a debate, “Free will means freedom from causation.” Other scientists who argue against free will say that it means that a soul or other supernatural entity causes behavior, and not surprisingly they consider such explanations unscientific. These arguments leave untouched the meaning of free will that most people understand, which is consciously making choices about what to do in the absence of external coercion, and accepting responsibility for one’s actions. Hardly anyone denies that people engage in logical reasoning and self-control to make choices. There is a genuine psychological reality behind the idea of free will. The debate is merely about whether this reality deserves to be called free will. Setting aside the semantic debate, let’s try to understand what that underlying reality is. © 2013 The Slate Group, LLC.
Keyword: Consciousness
Link ID: 18713 - Posted: 09.28.2013
by Megan Gannon, Live Science Deep in the cloud forests of Central America, two species of singing mice put on a high-pitched opera to mark their territory and stave off clashes, researchers discovered. Alston's singing mouse (Scotinomys teguina) and the Chiriqui singing mouse (S. xerampelinus) have overlapping lifestyles in the cloud forests of Costa Rica and Panama. But the tawny cousins seem to establish geographic boundaries so they can avoid competing with each other. "A long-standing question in biology is why some animals are found in particular places and not others," study researcher Bret Pasch, a postdoctoral fellow at the the University of Texas at Austin, said in a statement. "What factors govern the distribution of species across space?" As it turns out, a little communication between individuals affects the spread of both species as a whole. Both species of singing mice produce vocalizations that are barely audible to humans. As video footage of the mouse-y opera from the foggy forest floor shows, the creatures throw their heads back and belt out songs in the form of rapidly repeated notes, known as trills. The Alston's mouse in the clip even looks likes it's taking a bow after its solo. © 2013 Discovery Communications, LLC
Keyword: Aggression; Animal Communication
Link ID: 18712 - Posted: 09.28.2013